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Titlebook: Biology of Brain Dysfunction; Volume 3 Gerald E. Gaull Book 1975 Plenum Press, New York 1975 biology.brain.cortex.growth.information.molecu

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Zusammenfassung und Diskussion,e the potential of producing these defects as sequelae to intrauterine infections. This has stimulated efforts to understand their pathogenesis and to discover additional virus-host relationships existing within this significant group of diseases.
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Disorders of Organic Acid Metabolism,dentified by the ninhydrin reaction, and keto acids could be identified as dinitrophenylhydrozones. The “organic acids,” however, were difficult to detect by the then-existing methods. Because of these technical limitations, enzymatic defects that occur at later stages of amino acid metabolism were not discovered until 1966.
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Book 1975rain dysfunction. This explosion of exciting new information is crying out for collation and meaningful synthesis. In its totality, it defies systematic summa­ tion, and, of course, no one author can cope. Thus invitations for contributions were given to various experts in areas which are under acti
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nesis of brain dysfunction. This explosion of exciting new information is crying out for collation and meaningful synthesis. In its totality, it defies systematic summa­ tion, and, of course, no one author can cope. Thus invitations for contributions were given to various experts in areas which are
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Pathophysiology of Perinatal Hypoxic-ischemic Brain Damage,nown anatomical or chemical disorders, the causes for many of the cases remain elusive (Fig. 1). Among the known causes of mental and physical retardation, perinatal anoxic-ischemic injury ranks high, and we review here the present understanding and some of the known problems of this important cause of human disability.
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Pathogenesis of Brain Dysfunction in Deficiency of Thiamine, Riboflavin, Pantothenic Acid, or Vitamse vitamins are all involved in enzyme systems of the body as coenzymes, and a deficiency therefore results in multiple biochemical disturbances. We have tried to relate these biochemical disturbances, where possible, to the accompanying physical and neurological changes.
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https://doi.org/10.1007/978-1-4684-2673-1biology; brain; cortex; growth; information; molecular biology; physiology
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