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Titlebook: Biology of Brain Dysfunction; Volume 3 Gerald E. Gaull Book 1975 Plenum Press, New York 1975 biology.brain.cortex.growth.information.molecu

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发表于 2025-3-21 16:53:54 | 显示全部楼层 |阅读模式
期刊全称Biology of Brain Dysfunction
期刊简称Volume 3
影响因子2023Gerald E. Gaull
视频videohttp://file.papertrans.cn/188/187681/187681.mp4
图书封面Titlebook: Biology of Brain Dysfunction; Volume 3 Gerald E. Gaull Book 1975 Plenum Press, New York 1975 biology.brain.cortex.growth.information.molecu
影响因子The growth of neurochemistry. molecular biology, and biochemical genetics has led to a burgeoning of new information relevant to the pathogenesis of brain dysfunction. This explosion of exciting new information is crying out for collation and meaningful synthesis. In its totality, it defies systematic summa­ tion, and, of course, no one author can cope. Thus invitations for contributions were given to various experts in areas which are under active investigation, of current neurological interest, and pregnant. Although this project is relatively comprehensive, by dint of size. other topics might have been included; the selection was solely my responsibility. I believe systematic summation a virtual impossibility-indeed, hardly worth the effort. The attempt to assemble all of the sections involved in a large treatise with multiple authors inevitably results in untoward delays due to the difference in the rate at which various authors work. Therefore, the following strategy has been adopted: multiple small volumes and a relatively flexible format, with publication in order of receipt and as soon as enough chapters are assembled to make publication practical and economical. In this wa
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发表于 2025-3-21 23:54:37 | 显示全部楼层
Pathogenesis of Brain Dysfunction in Inborn Errors of Amino Acid Metabolism,ar association of an inherited disorder of amino acid metabolism with brain dysfunction. The precise identification by Jervis of the enzymatic etiology, the deficiency of Phenylalanine 4-hydroxylase (EC 1.14.16.1),. waited two decades. In the subsequent two decades a large number of inherited disord
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Disorders of Organic Acid Metabolism, acidemias were discovered. However, the inborn errors detected by amino acid chromatography were limited to those with defective enzymes in the first two steps of amino acid metabolism. In these cases, the parent amino acid or its keto analogue accumulates.. After the two early steps, namely transa
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Biological Aspects of Affective Psychoses,he assumption implicit in most of this work is that these illnesses are associated with disturbances in hypothalamic and limbic system functions and that an important role in these disturbances may be played by alterations in brain neu-rochemistry, particularly in the metabolism of the monaminergic
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Pathogenesis of Slow Infections of the Central Nervous System,1950s.. These diseases included rida (the Icelandic term for scrapie), visna, maedi, and pulmonary adenomatosis, and they were characterized by: (1) a long interval between infection and the initial clinical signs of disease, and (2) a protracted clinical course, usually ending in serious illness or
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Ionizing Radiations and the Nervous System,iated with this form of energy have focused the attention of both the scientific community as well as the public on an understanding of the biological effects of these radiations. The beneficial uses of atomic energy such as the development of nuclear power plants and the use of radiations in medica
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Brain Dysfunction in Congenital Malformations of the Nervous System,l data available in a number of surveys. (.. also Table 1). A more recent study conducted under the auspices of the World Health Organization and reported by Stevenson ... showed the total incidence of major congenital malformations in 411,405 births pooled from different surveys to be 12.7/1000 bir
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Pathogenesis of Brain Dysfunction in Deficiency of Thiamine, Riboflavin, Pantothenic Acid, or Vitamboflavin, pantothenic acid, and vitamin B.—on the mammalian system and how such deficiency affects the central nervous system and its functioning. These vitamins are all involved in enzyme systems of the body as coenzymes, and a deficiency therefore results in multiple biochemical disturbances. We h
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