Titlebook: Biology of Aging; Robert Zwilling,Cesare Balduini Book 1992 Springer-Verlag Berlin Heidelberg 1992 Alzheimer.alzheimer‘s disease.biology.b

瘙痒

Mitigate

Reihe der Villa Vigonihttp://image.papertrans.cn/b/image/187673.jpg

渐变

https://doi.org/10.1007/978-3-642-85789-8Alzheimer; alzheimer‘s disease; biology; brain; dementia; receptor

BATE

978-3-540-54488-3Springer-Verlag Berlin Heidelberg 1992

赏钱

https://doi.org/10.1007/978-3-211-89012-7us. Man has always followed with amazed curiosity and emotional interest the changes accompanying birth, development, aging, and death. And human beings date back to the very day when they became aware of aging and death and started to bury the dead. But superstition and ignorance also have always o

CAMEO

https://doi.org/10.1007/978-3-211-89012-7an accumulation of stochastic damage to DNA [16]. Damaged DNA is continuously repaired by several mechanisms in both proliferating and resting cells in order to avoid defective replication and abnormalities in transcription and translation [7]. One sort of such damage is the presence of uracil in DN

GROWL

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NICHE

https://doi.org/10.1007/978-3-642-76462-2ctorial translocation of protons across the inner membrane. This proton electrochemical potential (Δ.H .) drives ATP synthesis, ion translocation, and protein importation. At three steps along the electron transfer chain, represented by protein-lipid enzyme complexes I, III, and IV, oxidative energy

debris

M. W. Austen,J. M. Janas,H. R. Wiehlee. Several experimental models have been developed in a variety of animals to investigate the biology and pathophysiology of aging. Studies performed with them have alternatively provided evidence supporting a wide variety of different pathogenetic mechanisms. Recently, the so-called oxidative theor