Titlebook: Biochemistry of Apoptosis and Autophagy; Lorrie A. Kirshenbaum Book 2022 Springer Nature Switzerland AG 2022 Apoptosis.Autophagy.Cell Deat

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https://doi.org/10.1007/978-3-322-90036-4scarring if sufficiently excessive. Fibrosis affects many tissue types, and thus contributes to a broad group of diseases which, with few exceptions, continues to lack specific therapy. It has been estimated that nearly 45% of deaths in the developed world are caused by fibroproliferative diseases,

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Hans Joachim Salize,Wulf Rösslerbrosis, myocardial infarction, arrhythmias, and cardiomyopathies leading to cardiac dysfunction and heart failure remain significant contributors to this problem. For decades, therapeutic intervention has relied almost exclusively on surgical or pharmacologic approaches, and while these have improve

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Kosten und Leistungen von Baumaschinens review we discuss the interplay between mitochondria and the cells they occupy. Cells regulate the potentially lethal aspects of mitochondria by inserting both pro-apoptotic and anti-apoptotic machinery into mitochondria. In this way mitochondria can trigger apoptosis, ferroptosis, necrosis, and e

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Kosten und Leistungen von Baumaschinenxcess loss of cardiac myocytes and increased fibrosis, which leads to a reduced capacity to sustain contractile function. Cardiac myocytes are highly enriched in mitochondria which are responsible for generating energy via oxidative phosphorylation. Although mitochondria are critical for myocyte fun

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Berechnung der Durchschnittskosten particular, protein aggregation has been implicated in several neurodegenerative and cardiovascular diseases, such as Alzheimer’s disease and cardiac amyloidosis, that are associated with significant morbidity and mortality. Increasing evidence highlights that misfolded protein oligomers exert sign

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https://doi.org/10.1007/978-3-663-08128-9n made, there remain a need to identify additional strategies to manage heart disease and improve outcomes. Identifying the various signals and pathways that contribute to cardiac cell pathology and death, as well as those that promote cell survival and overall protection can lead to new ways to int

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