Titlebook: Biochemistry and Cell Biology of Ageing: Part III Biomedical Science; J. Robin Harris,Viktor I. Korolchuk Book 2023 The Editor(s) (if appl

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https://doi.org/10.1007/978-3-658-25196-3ells from murine embryos, to not only self-renew, but to also become all cells of the three germ layers of the developing embryo (Evans and Kaufman Nature 292:154-156, 1981), (Martin Proc Natl Acad Sci U S A 78:7634-7638, 1981). It took almost 20 years, following these discoveries, for this technolo

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0306-0225 and cutting-edge, this book is a valuable resource for experienced researchers and early career scientist alike, who are interested in learning more about the fascinating and challenging question of why and how our cells age..978-3-031-21412-7978-3-031-21410-3Series ISSN 0306-0225 Series E-ISSN 2542-8810

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Mitochondrial DNA Mutations and Ageing,t evidence surrounding the role of mitochondrial DNA mutations in the ageing process. It then focusses on more recent discoveries regarding the role of mitochondrial dysfunction in stem cell ageing and age-related inflammation.

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The Proteasome and Ageing,f damaged or misfolded proteins causes further reduction of proteasome activity. Abnormal proteins also accumulate as a result of age-related neurodegenerative diseases. Deficits in proteasome activity might be responsible for accumulation of protein aggregates and thus contribute to the pathology.

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Therapeutic Opportunities Presented by Modulation of Cellular Senescence,t mining have compiled co-mentions of cell senescence and age-related diseases allowing an impartial ranking of the impairments most closely associated with this process. Following this schema, the evidence for the involvement of senescence in several highly ranked pathologies is reviewed, alongside

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