Titlebook: Bioactive Heterocycles VI; Flavonoids and Antho Noboru Motohashi Book 2008 Springer-Verlag Berlin Heidelberg 2008 DNA.antioxidant.biochemis

初学者

Jealousy in Infants: Defended and Defined,against health damage by some types of harmful oxidants through different mechanisms. The purpose of this article is to review the biological activities of anthocyanins and the interactions of anthocyanins with DNA and protein.

inundate

Total Destruction: The Case of Jean Améry conductor-like screening model/parametric method 3 (COSMO/PM3) technique. The MIC values of phenothiazines [.–.] were well correlated to ΔΔ. ., HOMO energy and . .. QSAR may be applicable to predict the MIC of phenothiazines.

Interim

Functionality of Anthocyanins as Alternative Medicine,nge and apple and vegetables such as tomato, sweet pepper, spinachand radishes, and have been eaten as part of daily diets from the primitive era of human history. Whenanthocyanins are coupled with some water-soluble sugar molecules, they produce red, yellow, violet or bluecolors. This explains why

Ledger

Bioactive Mechanism of Interaction Between Anthocyanins and Macromolecules Like DNA and Proteins,getables and other plants. These are the glycosides of polyhydroxy and polymethoxy derivatives of 2-phenylbenzopyrylium or flavylium salts. The interest in anthocyanins has increased immensely during the past decade due to their array of healthily beneficial phytochemicals. Anthocyanins can protect

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Antibacterial Activity of Artificial Phenothiazines and Isoflavones from Plants,h Gram-positive and Gram-negative bacteria were equally sensitive, with the MIC varying between 25 and 100 μg/ml with most agents. Some phenothiazines were bactericidal, while others were bacteriostatic in action. Similar activity could be observed in isoflavonones obtained from the plants . spp. Tr

Mangle

Multidrug Resistance Reversal on Cancer Cells by Selected Carotenoids, Flavonoids and Anthocyanins,rtant final cause of therapeutic failure. Therefore, some compounds which inhibit the function of the MDR-efflux proteins may improve the cytotoxic action in cancer chemotherapy. The mechanism of action was believed to be a competition between their resistance modifiers and the cytotoxic agents for

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连累

Tumor Specificity and the Type of Cell Death Induced by Heterocycles,of new highly tumor-selective compounds. The screening of highly selective compounds should be performed before the identification of the type of cell death (either apoptosis, autophagy, or necrosis) and the cell death induction mechanism. The tumor specificities of heterocyclic compounds and the ty

骗子

Advanced Dihydropyridines as Novel Multidrug Resistance Modifiers and Reversing Agents,e this, these DHPs are significantly potent in cancer chemotherapy along with clinically used drugs. This family of compounds is potent inhibitors of P-glycoprotein (Pgp), which are the main cause of the efflux of toxins the cells. This review mainly focuses on the Pgp inhibitory property of DHPs. T

cancer

Theoretical Studies on Phenothiazines, Benzo[,]phenothiazines, and Benz[,]acridines,as investigated based on the theoretical calculations. Four different dipole moments (. ., . ., . ., and . .) and heats of formation (Δ. .) of the phenothiazines [.–.], benzo [.]phenothiazines [.–.], and benz[.]acridines [.–.] were separately calculated in the gas-phase and the water-solution by the