Titlebook: Basic Immunology and Its Clinical Application; Mitsuru Matsumoto Book 2024 The Editor(s) (if applicable) and The Author(s), under exclusiv

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季雨

Spinal-Tap

Orlando Musso,Bruno Clément,Nathalie Thérete variants in the AIRE gene and clinically manifests with multiple autoimmune diseases in patients. Studies on the function of the AIRE gene, discovered in 1997, have contributed to fundamental aspects of human immunology as they have been important in understanding the basic mechanism of immune bal

吝啬性

Stephen M. Riordan,Roger Williams, and some genetic factors. Because deficiency of Aire, a transcriptional regulator, is an autoimmune disease caused by a single gene abnormality, Aire is an ideal research target for approaching the enigma of autoimmunity, e.g., the mechanisms underlying Aire deficiency can be studied using genetic

强制令

Medicaid

Rajagopal N. Aravalli,Clifford J. Steerls are generally tolerant to self-antigens. However, when self-tolerance is broken, immune cells attack normal tissues or cells, leading to the development of autoimmune diseases. Genome-wide association studies have shown that MHC class II is the gene most strongly associated with the risk of most

pacifist

Novel Therapeutic Strategies to Combat HCC,lf-tolerance and homeostasis. Their functional or numerical anomalies can be causative of autoimmune and other inflammatory diseases. Recent advances in the research of the cellular and molecular basis of how Treg cells develop, exert suppression, and maintain their function have enabled devising va

侵略主义

Chemically-Induced Hepatocarcinogenesisders, but the underlying molecular or cellular mechanisms for the pathogenesis are often unclear. In order to clarify the pathogenesis, the mutant mice carrying the gene variants are apparently useful and important. Extensive analysis of those mice should contribute to the clarification of novel imm

漂亮才会豪华

MicroRNAs and Hepatocellular Carcinomals. NAs transported to the endosomal compartment are degraded by DNases and RNases. Degradation products, including single-stranded DNA, oligoRNA, and nucleosides, are recognized by TLR7, TLR8, and TLR9 to drive the defense responses against pathogens. NA degradation influences endosomal TLR respons

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