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Titlebook: Bacterial Toxins; Methods and Protocol Otto Holst Book 2000 Springer Science+Business Media New York 2000

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Margret Klinkhammer,Steffen Adler DNA, or proteins and lipids. Biospecific interaction analyses using SPR provides valuable information about the strength, speed, and stoichiometry of the interaction in real time and without the use of labels. An excellent review on the commercial SPR instrument called BIAcore™ has been published r
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https://doi.org/10.1007/978-3-322-91107-0which are among the most potent mitogens known for murine and human T lymphocytes (.,.). T-cell activation induced by SEs involves binding to constant parts of major histocompatibility complex (MHC) class II molecules and subsequent interaction with T cells expressing certain T-cell receptor (TCR) V
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Liviu Constantinescu-Simon (Lehrt)al proteins comprise a light (M. R~50) and a heavy (M. R~100) chain that are disulfide linked. In mammals, these proteins are the causative agents of two severe neuroparalytic diseases, botulism and tetanus. Botulism manifests as a flaccid muscle paralysis caused by a near irreversible and selective
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Josef von Stackelberg,Manfred Schmoch exotoxin, another toxin (.). In contrast to the secreted exotoxins this new, heat-stable toxin was found to be a constituent of the bacterial cell, and therefore Pfeiffer termed it endotoxin. Today we know that endotoxin (lipopolysaccha-ride, LPS) is the main outer membrane component of Gram-negati
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Manfred Schmoch,Dominik Steinerger a cascade of immunological responses in mammals including endotoxic effects and serum antibody production. LPSs have been found to exhibit a common molecular architecture consisting of at least two distinct regions: a carbohydrate-containing region and a lipid moiety referred to as lipid A (.).
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Auslegung eines Elektrofilters,lipid moiety, lipid A, which comprises a (phosphorylated) disaccharide of glucosamine or 2,3-diamino-2,3-dideoxy-D-glucose that is acylated by ester- and amide-bound fatty acids, and of the core region (.) which is covalently linked to lipid A. Only in S-form LPS is this core region substituted furt
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978-1-4899-4138-1Springer Science+Business Media New York 2000
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