Titlebook: Axoplasmic Transport in Physiology and Pathology; Dieter G. Weiss,Alfredo Gorio Conference proceedings 1982 Springer-Verlag Berlin Heidelb

mercenary

Disruption of Axoplasmic Transport by Neurotoxic Agents. The 2,5-Hexanedione Modelch as colchicine and the vinca alkaloids (e.g. vincristine, vinblastine). These compounds, together with podophyllotoxin and the maytansinoids (maytansine and maytanprine), possess a special affinity for tubulin, block the formation of axonal microtubules and, perhaps for these reasons, disrupt axon

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Axoplasmic Transport in the Experimental Neuropathy Induced by Acrylamidein these axonal enlargements (see review [13]). Pioneer investigations on axonal flow of labeled proteins have indicated that acrylamide intoxication interrupted slow axoplasmic transport in peripheral nerves, preferentially in sensory neurons [10]. However, the impairment of slow axoplasmic flow wa

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Disulfiram-Induced Impairment of Horseradish Peroxidase Retrograde Transport in Rat Sciatic Nerve axonal transport may play a role in the pathogenesis of peripheral neuropathies [3]. We studied the relation between impaired axonal transport and nerve damage using the disulfiram neuropathy model of the rat [1]. This animal model was developed as an experimental counterpart to the neuropathy obse

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Axoplasmic Transport and Synaptic TransmissionThe aim of this contribution primarily is to give a short review of studies which point to the close dependence on anterograde axonal transport of events, related to synaptic transmission.

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https://doi.org/10.1057/9780230288614only been established for a short time. A trans-synaptic transfer of radioactive proteins at neuromuscular junctions was first suggested, but not clearly demonstrated, by the low resolution autoradiographs of Korr et al. in 1967 [14]. In 1971 Bernice Grafstein demonstrated transfer of radioactivity