Titlebook: Applied Physiology to Reduce Ventilator Induced Lung Injury; Clinical Application Gary F. Nieman,Nader M. Habashi Book 2024 The Editor(s) (

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https://doi.org/10.1007/b138721ing the lung to a secondary ventilator-induced lung injury (VILI). In Chapters ., .–., a unifying hypothesis (Fig. .) on the mechanisms by which these mechanical alterations cause VILI-induced tissue damage was established. In addition, the current lung-protective ventilation strategies (Chaps. . an

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Book 2024necessary to preserve lung function. Mortality associated with the acute respiratory distress syndrome (ARDS), including that caused by COVID-19, remains unacceptably high. The primary treatment is supportive in the form of protective mechanical ventilation, but set improperly this can cause an unin

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science research into clinical practice.Identifies innovativ.This book details the mechanisms of ventilator induced lung injury (VILI) at the alveolar level with the aim to identify optimal ventilation methods necessary to preserve lung function. Mortality associated with the acute respiratory distr

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The Acutely Injured Lung Becomes Time/Press Dependent, with a functioning surfactant system, results in a very stable homogeneous inflation pattern (Chap. .). In the acutely injured lung, loss of these stabilizing factors renders the lung time- and pressure-dependent, meaning it will take more time for alveoli to open and less time for them to collapse