Titlebook: Adrenoceptors; Jillian G. Baker,Martin C. Michel,Roger J. Summers Book 2024 The Editor(s) (if applicable) and The Author(s), under exclusi

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Presynaptic Adrenoceptors,t by interrupting physiological autoinhibion of release. It belongs to the most effective antidepressive drugs. β.-Adrenoceptors are also localized on axon terminals in the peripheral and central nervous systems. Their activation leads to enhanced transmitter release, however, they are not activated

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,Roles of β-adrenoceptor Subtypes and Therapeutics in Human Cardiovascular Disease: Heart Failure, Tvariable blockade of β.-AR depending on relative affinity for β.-AR vs β.-AR. Since the introduction of β-blockers into clinical practice in 1965, β-blockers with different properties have been trialled, used and evaluated, leading to better understanding of their therapeutic effects and tolerabilit

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Adrenoceptors and Hypertension, longer first-line therapy after outcome trials did not demonstrate any benefits over and above other agents, despite similar blood pressure reductions. Angiotensin-converting enzyme inhibitors (or angiotensin receptor blockers), calcium channel blockers, and thiazide-like diuretics are now consider

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Locus Coeruleus and Noradrenergic Pharmacology in Neurodegenerative Disease,gic approaches are receiving attention in recent clinical studies: preserving noradrenaline levels (e.g., via reuptake inhibition) and direct activation of target adrenoceptors. Here, we review the expression and role of adrenoceptors in the brain, the preclinical studies which demonstrate that adre

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Adrenoceptor Expression and Function in the Endocrine Pancreas, factor for the development of type 2 diabetes. Thus, the α.-AR was proposed as a drug target for the treatment of some forms of type 2 diabetes. Drug research and development programs leveraging this mechanism have reached the clinical stage, but none have resulted in an approved medicine due to a

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