Titlebook: Autoimmunity; Andrzej Górski,Hubert Krotkiewski,Michał Zimecki Book 2001 Springer Science+Business Media New York 2001 Arthritis.Diabetes.

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https://doi.org/10.1007/978-3-319-62212-5. In patients with RA, proinflammatory cytokines secreted by synovial cells provoke local inflammation in the joints and, simultaneously, initiate a systemic acute phase response. Thus, profound changes of the neuro-endocrine-immune axis could take place in the patients. Defects in the hypothalamus-

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The Paradigms of Causality and Treatment for Autoimmune Disease,ll not cure the patient. This does not mean that effective treatments cannot emerge from faulty diagnoses. In truth, much of our therapeutic ensemble is composed of drugs developed as a result of chance observation, random screening, intuition or pre-scientific tradition. Nevertheless, the way to ef

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,A Role for NF-κB and the Proteasome in Autoimmunity,th a pre-diabetic state characterized by the production of autoantibodies specific for proteins expressed by pancreatic β cells. The non-obese diabetic (NOD) mouse is a spontaneous model of type 1 diabetes with a strong genetic component that maps to the major histocompatibility complex (MHC) region

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,Pathogenic Immunity in Theiler’s Virus-Induced Demyelinating Disease: A Viral Model for Multiple Scssociated with viral infection. The majority of experimental models rely heavily on the autoimmune components since similar diseases can be induced following immunization with various myelin antigens. A very attractive alternative model is the Theiler’s murine encephalomyelitis virus-induced demyeli