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Titlebook: Autoimmunity; Andrzej Górski,Hubert Krotkiewski,Michał Zimecki Book 2001 Springer Science+Business Media New York 2001 Arthritis.Diabetes.

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发表于 2025-3-21 18:14:08 | 显示全部楼层 |阅读模式
期刊全称Autoimmunity
影响因子2023Andrzej Górski,Hubert Krotkiewski,Michał Zimecki
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图书封面Titlebook: Autoimmunity;  Andrzej Górski,Hubert Krotkiewski,Michał Zimecki Book 2001 Springer Science+Business Media New York 2001 Arthritis.Diabetes.
Pindex Book 2001
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书目名称Autoimmunity影响因子(影响力)




书目名称Autoimmunity影响因子(影响力)学科排名




书目名称Autoimmunity网络公开度




书目名称Autoimmunity网络公开度学科排名




书目名称Autoimmunity被引频次




书目名称Autoimmunity被引频次学科排名




书目名称Autoimmunity年度引用




书目名称Autoimmunity年度引用学科排名




书目名称Autoimmunity读者反馈




书目名称Autoimmunity读者反馈学科排名




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Complement Regulatory Proteins and Autoimmunity,indings suggest that complement regulatory proteins have dual roles at inflammatory sites: enhancement of cellular resistance to complement attack and acceleration of theclearance of cells injurious to the organism due to complement-mediated mechanisms. To assist the former function, a therapeutic a
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Evidence that Fas and Fasl Contribute to the Pathogenesis of Experimental Autoimmune Encephalomyelirse can be acute or chronic relapsing, characterized by a rapid onset of hind limb weakness that commonly progresses to paralysis, followed by spontaneous remission starting 7–10 days after the initial appearance of symptoms. EAE can also be induced passively by the adoptive transfer of . activated
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Is Lack of Peripheral Tolerance Induction a Cause for Diabetes in the Non-Obese Diabetic Mouse?,ing naive T cells, the dendritic cells are also involved in generating central and peripheral tolerance to self molecules. In this process DC2 cells appear to be more important for the development of peripheral tolerance than DC1 cells. Besides abnormalities in the development of bone marrow-derived
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The Role of T Cells in Rheumatoid Arthritis,ed CD4.CD28. T cells is fundamentally changed and is shifted towards tissue-injurious capabilities. CD4.CD28. T cells are particularly important in patients with extra-articular manifestations of RA, where they may have a direct role in vascular injury. Understanding the mechanisms underlying the lo
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