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Titlebook: Atlas of Renal Pathology; R. A. Risdon,D. R. Turner Book 1980 Springer Science+Business Media Dordrecht 1980 pathology

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期刊全称Atlas of Renal Pathology
影响因子2023R. A. Risdon,D. R. Turner
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学科分类Current Histopathology
图书封面Titlebook: Atlas of Renal Pathology;  R. A. Risdon,D. R. Turner Book 1980 Springer Science+Business Media Dordrecht 1980 pathology
影响因子This book is intended as a practical bench manual phological abnormalities in renal diseases, and for the hospital pathologist who wishes to have where appropriate these have been illustrated. access to a simple informative account of renal Although the main emphasis is on the pathology, pathology, particularly for the interpretation of the relevant clinical aspects of the conditions cov­ percutaneous needle biopsy specimens. I n addition ered are included in recognition of the fact that we trust it will be valuable to physicians working renal disease is an area in which correlation of the in the field of renal disease, for whom the interpre­ clinical and histopathological findings is particularly tation of renal biopsy material is directly relevant to important in reaching an informed diagnosis. patient management. Whilst a comprehensive coverage more appro­ priate to a larger text has not been attempted, the Acknowledgements text has been planned to give an adequate account of the more important non-neoplastic disease pro­ We would like to thank the technical staff of the cesses and their pathological appearances in the Histopathology Laboratories of The London Hos­ kidney. Point
Pindex Book 1980
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Renal Cystic Disease,al cysts are acquired lesions. One or more renal cysts can be found at necropsy in about half of all subjects over 50 years of age, and their comparative rarity in children and young adults strongly supports their acquired nature. These ‘simple’ cysts appear to arise by local blockage of nephrons by
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Renal Infarction, Cortical Necrosis and Tubular Necrosis,ssels involved. Arterial occlusion is most frequently due to thromboembolism by thrombus formed within the heart cavities as a complication of myocardial infarction, atrial fibrillation or infective endocarditis. Occlusion may also be due to local causes within the renal arterial system such as athe
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Interstitial Nephritis,ish them from cases where there is no primary glomerular damage and the pathological changes affect predominantly the tubules and interstitium. For this purpose, the description ‘interstitial nephritis’ is used, and although it includes a variety of different diseases of known and unknown aetiology,
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,Diffuse Membranous Glomerulonephritis and ‘Minimal Change’ Disease,ellular proliferation (Figure 11.1). Some mesangial prominence is occasionally present. and in such cases the possibility of lupus nephritis should be excluded (see page 58). Electron microscopy, or the use of special techniques such as silver methenamine impregnation (Figure 11.2) or trichrome stai
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