Titlebook: Atlas of Renal Pathology; R. A. Risdon,D. R. Turner Book 1980 Springer Science+Business Media Dordrecht 1980 pathology

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Dorothee Horstkötter,Guido de Wertal cysts are acquired lesions. One or more renal cysts can be found at necropsy in about half of all subjects over 50 years of age, and their comparative rarity in children and young adults strongly supports their acquired nature. These ‘simple’ cysts appear to arise by local blockage of nephrons by

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4.5 Doses to Patients in Diagnostics,ish them from cases where there is no primary glomerular damage and the pathological changes affect predominantly the tubules and interstitium. For this purpose, the description ‘interstitial nephritis’ is used, and although it includes a variety of different diseases of known and unknown aetiology,

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https://doi.org/10.1007/978-3-642-23684-6he range of resulting morphological changes, and the functional abnormalities produced, provide the structural and clinical expressions of what is collectively termed glomerulonephritis. Such changes may be primary or part of a systemic disease, but the initial damage to the kidney occurs in the glo

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AGONY

4.2 Methods for Internal Dosimetry,arged and show prominent, lobulation. A variable proliferation of mesangial cells and increase in the amount of mesangial matrix is associated with diffuse thickening of glomerular capillary walls. This form of glomerulonephritis usually presents with an acute nephritic syndrome followed by a nephro

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4.2 Methods for Internal Dosimetry,ellular proliferation (Figure 11.1). Some mesangial prominence is occasionally present. and in such cases the possibility of lupus nephritis should be excluded (see page 58). Electron microscopy, or the use of special techniques such as silver methenamine impregnation (Figure 11.2) or trichrome stai

Affluence

4.5 Doses to Patients in Diagnostics,ent formation (Figures 13.1 and 13.2) and local scarring in some segmental proliferative lesions. Deposition of fibrin may be precipitated by antigen- antibody complexes whichare capable of releasing thrombogenic substances from platelets, and this probably explains its association with immunologica

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4.6 Doses to Patients in Therapy,ed, but is usually widely distributed throughout the body. Traditionally, generalized amyloidosis is classified as primary or secondary. Primary amyloidosis is either idiopathic or a complication of multiple myelomatosis; secondary amyloidosis follows a number of longstanding chronic inflammatory di