Titlebook: Atlas of Hypertension; Norman K. Hollenberg (Professor of Radiology, Dire Book 2003Latest edition Springer Science+Business Media New York

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https://doi.org/10.1007/978-3-642-65092-5though the existence of the renin-angiotensin system has been known for over two decades, recent advances in cell and molecular biology as well as renal physiology have opened the doors for a greater understanding of the role of this system in normal and disease states. Exciting new concepts, such a

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A. Everette James Jr.,S. Boyd Eaton Jr.muli of vascular constriction and on endocrine and renal stimuli that control blood volume. It has become clear that strong environmental and genetic influences converge to result in the hypertensive phenotype [1]. With the development of the science of vascular biology in recent years there has bee

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Die renale Ganzkorperclearance,ease. Hypertension, whether systolic, diastolic, or isolated systolic, is a major risk factor for vascular and target organ damage. Increased systolic blood pressure, increased pulse rate, and high baseline heart rate are definite cardiovascular risk factors. Recent data support the concept that eve

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https://doi.org/10.1007/3-540-28884-8rtery disease and renal disease have not been prevented [1,2]. Our ability to prevent hypertensive nephropathy through traditional methods of lowering BP may not be as effective as once thought, particularly in high-risk patients [3–6]. One reason for this may be our inability to adequately control