Titlebook: Atherosclerosis and Cardiovascular Diseases; Proceedings of the S S. Lenzi,G. C. Descovich Conference proceedings 1987 MTP Press Limited 19

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Atherosclerosis and ischaemic heart disease: surgical solutions 5%..Surgical results are analized in relation to the kind of operation (elective, urgent, emergency), the EF, the LVEDP, the number of distal anastomosis, the type of rivascularization (complete of incomplete) and the ventricular score.

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https://doi.org/10.1007/978-1-4612-3558-3ac occlusive atherosclerotic disease (Leriche’s syndrome) and predicted that excision of the diseased segment and its replacement with an aortic graft would be the ideal treatment for this condition. It was almost 30 years later, however, before this prediction became a reality when Oudot [3] performed the procedure for the first time (1951).

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Control-Group

Variational models for epitaxial growth,ue to the general trend towards a reduction in CHD which is manifest in the USA and many European countries and which in turn is due to a combination of social and environmental factors not yet clearly defined (2).

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Blowup Analysis for Stationary Solutions,atabolism of lipoproteins via the scavenger- or low affinity receptor pathway among others are causally linked with atherosclerosis, causing myocardial infarction (MI) peripheral vascular diseases (PVD) cerebrovascular diseases (CVD) and stroke.

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Social aspects of atherosclerosisue to the general trend towards a reduction in CHD which is manifest in the USA and many European countries and which in turn is due to a combination of social and environmental factors not yet clearly defined (2).

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Genetic basis of apolipoprotein disordersetabolism [4,5]. Conversely, apoC-III seems to inhibit the apoC-II activation of lipoprotein lipase [6]. It also modulates the uptake of apoE-containing lipoproteins by liver cells. ApoA-I [7] and possibly apoC-I [8], apoE [9] and apoA-IV [10] are thought to activate lecithin-cholesterol acyltransferase.

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Apolipoproteins in differential diagnosis of patients at high risk for atherosclerosisatabolism of lipoproteins via the scavenger- or low affinity receptor pathway among others are causally linked with atherosclerosis, causing myocardial infarction (MI) peripheral vascular diseases (PVD) cerebrovascular diseases (CVD) and stroke.