Titlebook: Atherosclerosis; Experimental Methods Angela F. Drew Book 2001 Humana Press 2001

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https://doi.org/10.1007/978-3-476-03666-7 agent, has been the standard imaging technique so far, in spite of its limitations. The severity of lumen-narrowing lesions is generally underestimated in X-ray angiography when compared to operative and histological findings, especially in cases of diffuse atherosclerosis or concentric stenosis. B

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https://doi.org/10.1007/978-3-531-92359-8ed macrophages, lymphocytes, and mast cells. Examining the role each cell type plays in the formation and pathophysiology of atherosclerotic plaques relies upon the identification of these cells within plaques and early plaque-like structures, which in turn depends on their expression of recognizabl

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https://doi.org/10.1007/978-3-663-14427-4, carotid, and peripheral vessels. Plasma Hcy can be considered a marker of methionine metabolic efficiency, mainly affected by dietary intake of vitamins, especially folate, vitamin B., and B., as well as by genetic mutations of key metabolic enzymes and renal elimination.

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https://doi.org/10.1007/978-3-322-80742-7nd qualitative histological analysis. A study design must include consideration of the parameters to be analyzed, to determine how tissue should be harvested. Often, studies increase the numbers of samples in each group to allow specimen collection by several techniques, and hence several different analyses can be performed.

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Palatial

Mechanical Injury Models,telet adhesion and degranulation, smooth muscle cell migration to the intima, and cell proliferation and matrix synthesis, ultimately producing a thickened neointimal layer. This model was developed initially by Baumgartner and Studer in the 1960s (.) and was modified (.) and used extensively throug