Titlebook: Arterial and Venous Systems in Essential Hypertension; M. E. Safar,G. M. London,Y. A. Weiss Book 1987 Martinus Nijhoff Publishers, Dordrec

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https://doi.org/10.1007/978-1-4899-0545-1iac hypertrophy in patients with chronic nephritis‘ … that it so affects the minute and capillary circulation, as to render greater action necessary to force the blood through the distant subdivisions of the vascular system.’ This point of view was emphasised by Sutton and by Gull [2], who in a lect

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https://doi.org/10.1007/978-1-4899-0545-1tor reflex. It is activated by the carotid and aortic baroreceptors and regulates heart rate, myocardial contractility, cardiac output, regional vasoactivity and blood flow distribution. However, this mechanism cannot counteract a permanent increase in pressure because the baroreceptors automaticall

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https://doi.org/10.1007/978-3-540-45316-1. The adequacy of the circulation depends on its filling pressure, especially on the filling pressure of the heart [1, 2]. For the overall circulatory system, a well-defined relationship must exist between changes in blood volume and changes in circulatory filling pressure [1]. The slope of the rela

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3 Electrode potentials of zero charge, in hypertension focused on arterial baroreceptors. However, with the recognition that cardiac mechanoreceptors contribute physiologically to the regulation of sympathetic nerve activity, renin release and vasopressin secretion, several investigators have evaluated the possibility that these recepto

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4 Free enthalpies of adsorption,ntake, both ECF and plasma volume are held constant. The ECF regulation implicitly expresses the concept that volume is not regulated per se but is controlled relative to the holding capacity of the vascular and interstitial spaces. In fact, ECF control is nearly similar to the control of circulator

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