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Titlebook: Arachidonic Acid Metabolism and Tumor Promotion; Susan M. Fischer,Thomas J. Slaga Book 1985 Martinus Nijhoff Publishing, Boston 1985 Nucle

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Esercizi di Termodinamica Applicatafor proteases in the regulation of normal cell growth, the functions of specialized cells, and in the control of tumor development, growth and metastases. The presence of a variety of protease inhibitors with varying types of specificity in plasma and cells of many tissues (8) points to a fine control of physiological functions for proteases.
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Circuiti idraulici industriali,gan systems, in turn, leads to adaptive responses on the whole organism level. Consequently, interference with any of these levels can influence the functions of levels above and below, because of interacting connections between the levels (1,2).
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Possible Involvement of Arachidonate Products in Tumor Promoter Inhibition of Cell-Cell Communicatigan systems, in turn, leads to adaptive responses on the whole organism level. Consequently, interference with any of these levels can influence the functions of levels above and below, because of interacting connections between the levels (1,2).
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Prostaglandins, Epidermal Hyperplasia and Skin Tumor Promotion,are modifiers of tissue response to that stimulus. The short half life of these compounds limits their activity to the site of production. As biologically potent locally acting compounds prostaglandins may play important regulatory roles in individual cell functions, cell interactions and tissue homeostasis.
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Effects of Phorbol Esters on Phospholipid Metabolism ,, localized within cell membranes (4,5) and one of the earliest and most striking consequences of phorbol ester action is the stimulation of membrane phospholipid metabolism. This has proven to be the case both with the in vivo mouse skin model (6–9) and with a variety of cell types in vitro.
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Proteases and Cyclic Nucleotides,for proteases in the regulation of normal cell growth, the functions of specialized cells, and in the control of tumor development, growth and metastases. The presence of a variety of protease inhibitors with varying types of specificity in plasma and cells of many tissues (8) points to a fine control of physiological functions for proteases.
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