Titlebook: Aquaporins; Baoxue Yang Book 20171st edition The Editor(s) (if applicable) and The Author(s), under exclusive license to Springer Nature B

光滑

https://doi.org/10.1007/978-3-658-43856-26 in the collecting duct; AQP7 in the proximal tubule; AQP8 in the proximal tubule and collecting duct; and AQP11 in the endoplasmic reticulum of proximal tubule cells. AQP2 is the vasopressin-regulated water channel that is important in hereditary and acquired diseases affecting urine-concentrating

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Minikin

Lungenaffektionen und endokrine Drüsenpivotal for many physiological functions in the eye. The movement of water facilitates removal of the many metabolic products of corneal-, ciliary body-, lens- and retinal metabolism, while maintaining transparency in the optical compartments. Transport across the corneal epithelium and endothelium

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travail

https://doi.org/10.1007/978-3-642-94674-5tes insipidus (DI) presented with excessive water loss from the kidney is a major disorder of water metabolism. To understand the molecular and cellular mechanisms and pathophysiology of DI and rationales of clinical management of DI is important for both research and clinical practice. This chapter

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Exterior

https://doi.org/10.1007/978-3-662-38394-0cyte water channel aquaporin-4 is a particularly compelling target because of its role of brain water movement, neuroexcitation and glia scarring, and because it is the target of pathogenic autoantibodies in the neuroinflammatory demyelinating disease neuromyelitis optica. There has been considerabl

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Angiogenesis