Titlebook: Applied Cardiovascular Physiology; Michael R. Pinsky Book 1997 Springer-Verlag Berlin Heidelberg 1997 Sepsis.cardiovascular function.cardi

CANE

Physiology of VO2/DO2aerobic metabolism takes over, leading to lactate production and acidosis. Cellular processes fail as energy supplies fall, and organ system function deteriorates. Oxygen is thus essential for the normal activity and survival of tissues.

resilience

0933-6788 hours in each day to the application of physiological principles to the bedside care of critically ill patients. The universal challenge of car­ diovascular instability confronts all health care providers who treat patients in an acute care setting. Whether that be in the field or Emergency Departme

prick-test

Social Cognition in Spectating,c interventions to such patients. This chapter will review the basic pathophysiology of cardiac failure, identify cellular targets for pharmacologic intervention, and discuss the actions and uses of some of the newer inotropic agents.

micronutrients

venous-leak

悬挂

Left Ventricular Afterload and Ventriculo-Arterial Couplingtudy of the energy transfer between a source and the load attached to it, receiving a part of that energy [1]. The ideal coupling is achieved when the maximum of energy is transferred by the source to the load, with minimal waste in friction and heat. In the case of the heart and the systemic circul

Generic-Drug

华而不实

人工制品

immunity

Heart-Lung Interactionsprimary role of critical care resuscitative efforts is to insure the adequacy of O. delivery in stress states, an understanding of cardio-pulmonary physiology and the effects of disease and therapeutic interventions on cardio-pulmonary status is central to the management of the critically ill patien