Titlebook: Antioxidants and Cardiovascular Disease; Jean-Claude Tardif,Martial G. Bourassa Book 2000 Springer Science+Business Media Dordrecht 2000 L

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https://doi.org/10.1007/0-306-46940-5ls of serum malondialdehyde, a product of lipid peroxidation, can be correlated with severity of disease. In this review, the basic mechanisms by which free radicals effect cell injury and the potential role for compounds with antioxidant activity to intervene in these disease processes will be discussed.

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S. Durand-Vidal,J. -P. Simonin,P. Turq these patients. This chapter briefly reviews compensatory neurohumoral mechanisms as well as mechanisms potentially related to inflammation, apoptosis and oxidative stress in chronic congestive heart failure. Secondly, the presumed clinical benefits of antioxidant therapy in patients with CHF are reviewed.

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0166-9842 cyclooxygenase activation are all essential processes of life, which also generate oxidative species. In humans, chronic oxidative stress often coupled with deficiency of antioxidant defenses is associated with the aging process and can lead to the development of disorders such as cancer and arteria

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https://doi.org/10.1007/0-306-46940-5xidation (redox) reactions that generate reactive oxygen species (ROS) have gained attention as important chemical processes that regulate signal transduction. Accumulating evidence indicates that excessive ROS may be pathogenic for a wide variety of human diseases.

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Reactive Oxygen Species as Mediators of Signal Transduction in Cardiovascular Disease,xidation (redox) reactions that generate reactive oxygen species (ROS) have gained attention as important chemical processes that regulate signal transduction. Accumulating evidence indicates that excessive ROS may be pathogenic for a wide variety of human diseases.

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