Titlebook: Antiglobulins, cryoglobulins and glomerulonephritis; Second International Claudio Ponticelli,Luigi Minetti,Giuseppo D’Amico Book 1986 Marti

Nutrient

unopathophysiology of cryoimmunoglobulins and auto-antibodies to immunoglobulins (lg) and, secondarily, to explore the possible participation of these disordered states in glomerular injury. As such, this conference is principally devoted to an analysis of two properties of certain species of the globulin fra978-94-010-8406-2978-94-009-4289-9

nitric-oxide

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avulsion

Antiglobulins, cryoglobulins and glomerulonephritis978-94-009-4289-9

健忘症

rheumatology

Subsets of rheumatoid factors determined by cross idiotypesyoglobulinemia (Kunkel, 1973). Two distinct idiotype groups were described, a major group WA comprising approximately 60% of those proteins and a minor group PO comprising 20%; 20% were uncharacterized. More recently a third rheumatoid factor cross idiotype has been defined, the Bla group, that is d

assail

Structure and function of rheumatoid factor: implications for its role in the pathogenesis of mixed arthritis [1,2], they occur in a wide range of pathological and non-pathological states, and it soon became evident that RF were not specific for rheumatoid arthritis. Early studies demonstrated the regular appearance of RF during the course of chronic infections such as subacute bacterial endocardi

令人悲伤

Regulatory Idiotopes and Diseaseslymphocytes. Two categories of idiotopes are expressed on the immune receptor of lymphocytes. Individual idiotypes are clonotypic antigenic determinants expressed only by a single clone. These idiotypes results from somatic recombination or somatic mutational events. Generally, they are not inherite

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Pathogenetic mechanisms of infective endocarditis glomerulonephritis in the rabbitfrom them. Despite the fact, however, that some of them appear to be mediated by immune complex reactions, there is a paucity of models which have a close counterpart in human disease. (This is due partly to our lack of knowledge of the aetiology of the majority of human types of glomerulonephritis)

GRATE

Interaction of Human IgM Rheumatoid Factors with Immune Complexes in Experimental Murine Glomeruloneich immune complexes (IC’s) are felt to have a pathogenetic role. These diseases include bacterial endocarditis, several forms of glomerulonephritis, systemic lupus, vasculitis and cryoglobulinemia. In all these diseases rheumatoid factor (RF) occurs not only in the circulation, but has been identif