Titlebook: Animal Models of Acute Neurological Injuries II; Injury and Mechanist Jun Chen,Xiao-Ming Xu,John H. Zhang Book 2012 Springer Science+Busine

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书目名称Animal Models of Acute Neurological Injuries II影响因子(影响力)学科排名




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书目名称Animal Models of Acute Neurological Injuries II网络公开度学科排名




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书目名称Animal Models of Acute Neurological Injuries II被引频次学科排名




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书目名称Animal Models of Acute Neurological Injuries II读者反馈学科排名

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The Stereology and 3D Volume Analyses in Nervous Tissuee or features of a brain region can be analyzed with systematic computation. This chapter does not attempt to provide theoretical derivation, nor exhaust all infrequently used methods. Instead, practical uses and caveats are provided followed by the actual practice of the quantitation of commonly en

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EEG, Evoked Potential, and Extracellular Single-Unit Recordings In Vivoconnect with other neurons. Each neuron can potentially connect with many other neurons and vice versa. At the sites of connections, information is transmitted across synapses, neurotransmitters are released from presynaptic terminals, and these diffuse to receptor molecules located on the postsynap

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Intracellular Recording In Vivo and Patch-Clamp Recording on Brain Slicesological conditions, we need to know how the neuron works by monitoring the electrical activities of the neuron at different circumstances. Many electrophysiological techniques are available to obtain information from nerve system. This chapter introduces two techniques that can collect information

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Electrophysiological Evaluation of Synaptic Plasticity in Injured CNSrning and memory deficit and epilepsy. Field potential recording (FPR) and whole-cell patch clamp recording are electrophysiological techniques frequently used for evaluating synaptic plasticity. The FPR technique can measure synaptic activity generated from a population of activated neurons while p

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Characterization of RNA disease and prediction of outcome, as well as support pharmacogenomic approaches to novel treatment strategies. Variation of gene expression is an integral mechanism underlying susceptibility to complex disease and is directly modified by polymorphisms in regulatory elements. Thus, the study of gen

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