Titlebook: Angiotensin and the Heart; H. Grobecker,Gerd Heusch,B. E. Strauer Book 1993 Dr. Dietrich Steinkopff Verlag GmbH & Co. KG, Darmstadt 1993 A

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Myocardial perfusion dependent and independent mechanisms of regional myocardial dysfunction in hypeicular (LV) function, it could be of greater importance during periods of stress such as occurs during exercise where increased metabolic demands induced by the stress may not be fully met by increases in coronary blood flow. The impaired subendocardial coronary reserve is caused not only by the hyp

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ACE-inhibitors and coronary microcirculation patients with arterial hypertension often have symptoms of angina pectoris and a positive exercise tolerance test. The angina pectoris-symptoms in patients with arterial hypertension are due to functional and structural alterations of the coronary microcirculation. Consequently, an antihypertensive

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“Cardioprotection” by ACE-inhibitors in acute myocardial ischemia and infarction?nd positive inotropic agent. This has raised the possibility that angiotensin converting enzyme (ACE) inhibitors might be “cardioprotective” (that is, might attenuate myocardial injury, dysfunction and necrosis) in the setting of acute ischemia and infarction. Captopril, enalapril and ramipril have,

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Angiotensin-converting enzyme inhibitor in a human model of restenosisass grafting (CABG) to restore coronary blood flow in patients with obstructive coronary artery disease (15). Increased experience and advances in technology have resulted in a high primary success rate (over 90%) and a low compilation rate (death, non-fatal myocardial infarction; 4–5%) (9). However

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Angiotensin-converting enzyme inhibitors in the treatment of clinical heart failuree, there is convincing evidence that they improve survival and that, in this respect, they are superior to ordinary vasodilators. ACE-I administration also improves New York Heart Association functional class and the left-ventricular function. But their long-term effects on exercise tolerance and qu

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Basic aspects the cardiac renin-angiotensin system has been made. It is now accepted that the renin-angiotensin system originally detected as a classical humoral system, which secretes active molecules from a gland reaching their receptors of a target organ via the blood, may also reside in many tissues, especia