Titlebook: Angiogenesis; From the Molecular t Michael E. Maragoudakis Book 2000 The Editor(s) (if applicable) and The Author(s), under exclusive licen

morphology

书目名称Angiogenesis影响因子(影响力)




书目名称Angiogenesis影响因子(影响力)学科排名




书目名称Angiogenesis网络公开度




书目名称Angiogenesis网络公开度学科排名




书目名称Angiogenesis被引频次




书目名称Angiogenesis被引频次学科排名




书目名称Angiogenesis年度引用




书目名称Angiogenesis年度引用学科排名




书目名称Angiogenesis读者反馈




书目名称Angiogenesis读者反馈学科排名

乱砍

Tie-1 Receptor Tyrosine Kinase Endodomain Interaction with SHP2: Potential Signalling Mechanisms andendodomain having a ligand-independent signalling role in the endothelial cell. The potential roles of ectodomain cleavage and cleavage activated signalling in regulating microvessel stability in angiogenesis, vessel remodelling and regression are considered.

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On the Mechanism(s) of Thrombin Induced Angiogenesisothelial cells increases the vectorial secretion of extracellular matrix proteins, a process essential at the final steps of angiogenesis. In addition, thrombin potentiates the VEGF-induced mitogenesis of endothelial cells. This can be explained by the upregulation of the VEGF receptors (KDR & flt-1

Adjourn

Endothelial Receptor Tyrosine Kinases involved in Blood Vessel Development and Tumor Angiogenesists of tissues, and is triggered by hypoxia. Deregulated angiogenesis plays an important role in the pathogenesis of a variety of diseases, including retinopathy and solid tumor growth (Folkman, 1995).

Tracheotomy

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Signal Transduction and Transcriptional Regulation of Angiogenesiss of these cell lines with a parental murine EC line, MSS31. The growth rate was almost identical among three cell lines. It appeared that gene expressions of matrix metalloproteinases (MMP-1, MMP-3, and MMP-9) as well as integrin beta3 were correlated with the level of Ets-1 expression. As a result

神圣不可

Differential Contribution of Bradykinin Receptors in Angiogenesis inhibition in CVEC prevented BK/B1 induced cell growth. These results substantiate the protective and trophic role of BK on microvascular endothelium by demonstrating that Bl receptor stimulation on venular endothelium is mandatory for the angiogenic effect of BK through its coupling to NOS activat

可行

Sensitivity of Different Vascular Beds in the Eye to Neovascularization and Blood-Retinal Barrier Brvessels, and electron microscopy. Levels of transgene expression were determined by the polymerase chain reaction. In normal mice, retinal vessels are organized into a superficial and a deep capillary bed with some vessels forming a shunt between both beds. In a transgenic line of mice that overexpr

Prosaic

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