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Titlebook: Alzheimer’s Disease. Epidemiology, Neuropathology, Neurochemistry, and Clinics; Konrad Maurer,Peter Riederer,Helmut Beckmann Book 1990 Spr

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楼主: Ferret
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Quantitative investigations of presenile and senile changes of the human entorhinal regionIn a quantitative study on thick frozen sections stained by the Gallyas’ silver impregnation method neurofibrillary tangles (NFT), neuritic plaques (NP) and neuropil threads (NT) in the entorhinal as well as transentorhinal area (Braak and Braak, 1985) have been evaluated. The amount of NT exceeded by far that of NFT and NP.
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Descriptive and analytic epidemiology of Alzheimer’s disease for Alzheimer’s disease are concerned, our knowledges principally base on findings derived from case-control studies. A family history of dementia and a previous head trauma were found to be associated with Alzheimer’s disease in case-control studies. Indications of the role of genetic factors in t
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Morphology of Alzheimer’s disease and related disordersrtical atrophy with 40 to 60% loss of large neurons and 45 to 55% decline of synaptic density in frontal, temporal, parietal cortex and hippocampus are probable correlates of progressive dementia. Many mediator specific brainstem nuclei show progressive neuronal loss and presence of NFT with severe
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Die spezifisch antirachitischen Verfahren, for Alzheimer’s disease are concerned, our knowledges principally base on findings derived from case-control studies. A family history of dementia and a previous head trauma were found to be associated with Alzheimer’s disease in case-control studies. Indications of the role of genetic factors in t
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Die Behandlung von Gelenkknorpeldefektenrtical atrophy with 40 to 60% loss of large neurons and 45 to 55% decline of synaptic density in frontal, temporal, parietal cortex and hippocampus are probable correlates of progressive dementia. Many mediator specific brainstem nuclei show progressive neuronal loss and presence of NFT with severe
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Wissenschaftliche Taschenbücherer number of amyloid deposits. With age, the number of plaques decreased in superficial layers of the cerebral cortex (II–III) and increased in the deeper layers (IV–VI). In the oldest animals, diffuse amyloid deposits in the deeper layers of the cortex predominated. Our studies suggest that the fre
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