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Titlebook: Alcohol and Hormones; Ronald R. Watson Book 1995 Springer Science+Business Media New York 1995 growth.growth factor.growth hormone.homeost

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发表于 2025-3-21 16:25:27 | 显示全部楼层 |阅读模式
期刊全称Alcohol and Hormones
影响因子2023Ronald R. Watson
视频video
学科分类Drug and Alcohol Abuse Reviews
图书封面Titlebook: Alcohol and Hormones;  Ronald R. Watson Book 1995 Springer Science+Business Media New York 1995 growth.growth factor.growth hormone.homeost
影响因子Alcoholism, the oldest addictive illness, remains a serious problem because of its major modifications of many physiological systems. The genetic and neurological origins and mechanisms are complex and only partly understood. However, it is clear that alcohol use modifies the production of most hormones, directly and indirectly modulating the systems that hormones regulate. Acute and chronic alcohol consumption affects virtually all cellular functions of tissues like brain, liver, immune system, and reproductive and endocrine organs. The latter have major regula­ tory roles, affecting many functions from development to repro­ duction. Although stress can further modify alcohol‘s effects on hormone production, it significantly causes most of alcohol‘s modi­ fications of the body to occur via changes in hormone production and secretion. Ronald R. Watson vii Contributors Michael L. Adams • Department of Psychiatry, Washington University School of Medicine, St. Louis, MO Robert A. Anderson, Jr .• Ob/Gyn Research, Rush-Presbyterian-St. Luke‘s Medical Center, Chicago,IL Kasibhatla Bhavani • Molecular Heptology Laboratory, Massachusetts General Hospital Cancer Center, Harvard Medical Scho
Pindex Book 1995
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Neuroendocrine Responses to Ethanol in the Prepubertal Female Rat,nervous system plays a crucial role in bringing together these events by controlling both anterior pituitary function via the secretion of hypothalamic neuromodulators, and the ovary via pituitary hormone secretions and direct neural inputs. This being the case, a drug that is capable of affecting a
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The Effect of Ethanol on Male Rodent Reproduction and Growth,hanisms responsible for this clinical picture been elucidated. Although direct gonadal toxicity after ethanol (ETOH) exposure has been well documented, the failure of serum luteinizing hormone (LH) and serum follicle-stimulating hormone (FSH) levels to rise in the presence of blunted testosterone va
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Thyrotropin-Releasing Hormone (TRH),biological) and experiential/environmental factors.. As more is learned about alcoholism, it is clear that an indeterminate number of distinct and separate subgroups of the illness exist.. Advances in the understanding of the transmission of alcoholism and its heterogeneity have not as yet been matc
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Hidden Hormones in Alcoholic Beverages,alcoholic beverages. Now, mentally disassociate these endocrine effects from an alcohol etiology, and try to imagine an entirely different causal agent. Picture a quintessential alcoholic compensated-cirrhotic male with all extremes of endocrinopathy. He is both hypogonadal and feminized. He has gyn
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Gene Structure and Multiple Regulations of the Ethanol-Inducible Cytochrome P45O2E1 (CYP2E1) Subfamimmune system, reproductive and endocrine organs, and so forth.. In liver, ethanol drinking causes alcoholic fatty liver, hepatitis, fibrosis, and cirrhosis. Although the pathoetiological mechanisms of these disease states caused by ethanol consumption are still unknown, several mechanisms have been
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Thermoregulation and Alcohol,mperature,.owing to both metabolic depression.and augmented peripheral heat loss.. Likewise, it has been recognized that alcohol diminishes an individual’s capacity to maintain thermostability during exposure to extreme ambient temperatures,. and produces a paradoxical feeling of warmth with accompa
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