Titlebook: Advances in the Immunopathogenesis of Multiple Sclerosis; D. Gambi,P. A. Muraro,U. Ecari Conference proceedings 1999 Springer-Verlag Itali

懒惰人民

Rapid Reviews in Software Engineeringdily recognized as markers of infection by the innate immune system that instructs the adaptive response accordingly [2]. Based on this evidence, it is now clear that the correct functioning of innate and adaptive immunity as well as their balanced interaction are essential for a physiological immun

Demulcent

Catrien Notermans,Maya Turolla,Willy Jansendentified components (i.e. oligoclonal cerebrospinal fluid bands), and γ/δ T cells represent the effector cell population. Nevertheless, the two different cell populations display overlapping functions; a minor proportion of α/β T cells specific for myelin antigens shows cytotoxic properties while γ

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郊外

The role of proinflammatory cytokines in multiple sclerosis,dentified components (i.e. oligoclonal cerebrospinal fluid bands), and γ/δ T cells represent the effector cell population. Nevertheless, the two different cell populations display overlapping functions; a minor proportion of α/β T cells specific for myelin antigens shows cytotoxic properties while γ

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NOVA

,The naïve and memory MBP-reactive CD4+ T cell repertoire: implications for the autoimmune concept iof MS is currently ascribed, at least in part, to a T cell-mediated process targeting myelin components [1].Among myelin proteins, myelin basic protein (MBP) is a major candidate autoantigen in MS. The evidence supporting this claim has been examined in several excellent reviews [1–4], and includes

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blithe

Immune activation in the interface between innate immunity and adaptive response:in vitro studies a level of the adaptive immune response, with particular emphasis on the search for potential T lymphocyte autoantigens. These investigations have been, in many cases, unrewarding. One reason may be that T cells are not the key players in self-nonself discrimination: T cell receptors (TCR) have rando

immunity

Neuronal control of the immunological microenvironment in the CNS: implications on neuronal cell deles are virtually absent. Heterodimeric MHC molecules are essential for the initiation, propagation and effector phases of antigen-specific immune responses. Endogenous and exogenous antigenic peptides are presented via MHC molecules to T lymphocytes to enable cognate interactions. While MHC molecul

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Levels of platelet-activating factor in cerebrospinal fluid and plasma of patients with relapsing-rough a seven-spanning transmembrane domain receptor [1, 2]. Following appropriate stimulation, it is produced and released by monocytes, neutrophils, endothelial cells and T lymphocytes [3–8]. It is also produced by neurons and glial cells stimulated by neurotransmitters and tumor necrosis factor (T