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Titlebook: Advances in Research on Neurodegeneration; Volume 6 W. Poewe,G. Ransmayr Conference proceedings 1999 Springer-Verlag Wien 1999 Huntington‘s

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Neural transplantation in animal models of multiple system atrophy: a review,opamine (6OHDA) and quinolinic acid (QA) into medial forebrain bundle and ipsilateral striatum, respectively (“double toxin — double lesion” approach). Preliminary evidence in rodents subjected to such lesions indicates that embryonic transplantation may partially reverse behavioural abnormalities.
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https://doi.org/10.1007/978-3-642-35401-4han bromocriptine in this indication, at the cost of very similar adverse effects..In de novo levodopa naive patients, pramipexole and ropinirole did significantly better than placebo in short-term (few months) follow-up trials, at the cost again of classical dopaminergic adverse effects. Ropinirole
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Multidimensional Diffusion Models during incubation; CHO-K1 cells exposed to apomorphine for a period as short as 1 h and then allowed to grow for three days were significantly reduced in number with respect to untreated control cells. After four hours of exposition to apomorphine (10 μM) the antiproliferative effect was similar to
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Multidimensional Diffusion Modelstyrosine hydroxylase activity. It is suggested that the neuroprotective effect of (R)-apomorphine against MPTP neurotoxicity derives from its radical scavenging and MAO inhibitory actions and not from its agonistic activity, since the mechanism of MPTP dopaminergic neurotoxicity involves the generat
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P. Dräxler,C. M. Ringel,G. O. Michleropamine (6OHDA) and quinolinic acid (QA) into medial forebrain bundle and ipsilateral striatum, respectively (“double toxin — double lesion” approach). Preliminary evidence in rodents subjected to such lesions indicates that embryonic transplantation may partially reverse behavioural abnormalities.
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0303-6995 Overview: 978-3-211-83261-5978-3-7091-6369-6Series ISSN 0303-6995
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Journal of Neural Transmission. Supplementahttp://image.papertrans.cn/a/image/149582.jpg
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