Titlebook: Advances in Research on Neurodegeneration; Volume 5 P. Riederer,D. B. Calne,M. B. H. Youdim Conference proceedings 1997 Springer-Verlag Wie

新义

Induction of mitosis-related genes during dopamine-triggered apoptosis in sympathetic neuronsmed cell death in both neuronal and non-neuronal cell types. We examined the molecular changes associated with dopamine-triggered apoptosis in sympathetic neurons using the differential display approach, and isolated 14 different DA responsive genes whose expression is altered during the early stage

aesthetic

Neuronal vulnerability in Parkinson’s diseases disease. This suggests that some specific factors make subpopulations of dopaminergic neurons more susceptible to the disease. Here, we show that the most vulnerable neurons are particularly sensitive to oxidative stress and rise in intracellular calcium concentrations. Because both events seem to

HARP

theta-waves

The halogenated tetrahydro-β-carboline “TaClo”: A progressively-acting neurotoxin, increases the sensitivity to apomorphine in aging rats after subchronic daily application (0.2 mg/kg i.p.) for 7 weeks. Nine weeks after the last treatment with TaClo, the running speed of the animals during spontaneous nocturnal activity was significantly diminished, and 9 months after the last i

cauda-equina

Developmental and genetic regulation of programmed neuronal deathThe principal action of neurotrophins such as nerve growth factor is probably the suppression of the preexistent machinery of programmed cell death that is readily activated in neurons deprived of neurotrophins. Potassium-mediated neuronal depolarization prolongs neuronal survival in vitro and has b

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