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Titlebook: Advances in Myocardiology; Volume 4 E. Chazov,V. Saks,G. Rona Book 1983 Springer Science+Business Media New York 1983 cardiology.heart.hear

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楼主: estrange
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https://doi.org/10.1007/3-540-53031-2oxidation by antioxidants, inhibition of proteolytic lysosomal enzymes with trasilol, or blocking of calcium entry into the myocytes with verapamil. The possible application to clinical practice of chemical prophylaxis of stress damage in the heart is discussed.
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Free partialy commutative monoids,m level at terminal stage, suggesting that the NE turnover is related to the progression of the disease. In light of the present findings, it can be surmised that NE plays a permissive role in the genesis of the hamster disease by promoting the heart necrotic changes.
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Valérie Berthé,Herman Goulet-Ouelletase in heart weight to more than 160% of controls corresponds to [.H]thymidine labeling of 38.06 ± 4.65 and 21.67 ± 4.16% in left and right atrial myocytes, respectively, whereas in hearts weighing less than 140% of controls, [.H]thymidine labels only 8.20 ± 1.93% in the left atrium and 3.94 ± 1.57%
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