Titlebook: Advances in Down Syndrome Research; G. Lubec Book 2003 Springer-Verlag/Wien 2003 Alzheimer‘s disease.Chromosom.Down syndrome.Emotion.Micro

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书目名称Advances in Down Syndrome Research影响因子(影响力)




书目名称Advances in Down Syndrome Research影响因子(影响力)学科排名




书目名称Advances in Down Syndrome Research网络公开度




书目名称Advances in Down Syndrome Research网络公开度学科排名




书目名称Advances in Down Syndrome Research被引频次




书目名称Advances in Down Syndrome Research被引频次学科排名




书目名称Advances in Down Syndrome Research年度引用




书目名称Advances in Down Syndrome Research年度引用学科排名




书目名称Advances in Down Syndrome Research读者反馈




书目名称Advances in Down Syndrome Research读者反馈学科排名

Frequency

Aberrant protein expression of transcription factors BACH1 and ERG, both encoded on chromosome 21, ve received increasing importance as core vehicles for strategy-making. These coordinated undertakings enable firms to reinforce the current strategy or alter it in order to realign the organization in accordance with changed environmental conditions. Despite the growing importance of strategic init

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magnate

Transcription factor REST dependent proteins are comparable between Down Syndrome and control brain of public sector pension systems in OECD countries. Thecountries covered are Denmark, Finland, Germany, The Netherlands,Norway, and the USA. Part II considers the regulation of occupationalpension schemes in The Netherlands and the United Kingdom, and whetherthere is still a role for the State in p

Mindfulness

Chromosome 21 KIR channels in brain development,ariable approach.Provides answers to China’s economic and po.This book examines China’s economic development from the end of 1970s, integrating perspectives from law, economics and political science. Particular attention is given to the role of formal law and political changes in China’s development

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G. LubecIncludes supplementary material:

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Journal of Neural Transmission. Supplementahttp://image.papertrans.cn/a/image/147861.jpg

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内向者

Schlussfolgerungen und Empfehlungen,uent cause of congenital heart defects and the leading genetic cause of mental retardation. Although overexpression of a gene(s) or gene cluster on human chromosome 21 (Chr 21) or the genome imbalance by Ts21 has been suggested to play a key role in bringing about the diverse DS phenotypes, little i