Titlebook: Adenosine, Cardioprotection and Its Clinical Application; Robert M. Mentzer,Masafumi Kitakaze,Masatsugu Hori Book 1997 Kluwer Academic Pub

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Wiederholungsfragen mit Antworten, coronary perfusion pressure, myocardial contractility decreases, recognized as Gregg ’s phenomenon. There are several hypotheses to explain this phenomenon: 1) decreases in sarcomere length of the myofilaments, 2) reversal of latent myocardial ischemia, 3) release of cardiodepressive agents, and 4)

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https://doi.org/10.1007/b137967dioprotection have become more intriguing, and more complex. Adenosine has potential to exert cardioprotection during all three windows of cardioprotection (pretreatment or preconditioning, ischemia and reperfusion). Therefore, adenosine has emerged as a broad-spectrum cardioprotective agent that ex

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Grundlagen und Voraussetzungen,ct of ischemic preconditioning. Indeed, it has been clarified that adenosine contributes to limit infarct size.. There are two different possibilities by which adenosine links with cardioprotection in ischemic preconditioning. One possibility is that release of adenosine during ischemia and reperfus

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https://doi.org/10.1007/b137967s would be expected to result in a decreased rate of calcium influx via voltage-sensitive calcium channels and sodium-calcium exchange and reduce calcium overload and lethal cell injury if timely reperfusion were to occur. A number of studies support this hypothesis, although several recent reports

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https://doi.org/10.1007/b137967 protect the heart against ischemia/reperfusion injury, including arrhythmia, contractile dysfunction and necrosis. Secondly, bradykinin may play a role in an endogenous cardioprotective mechanism, ischemic preconditioning (i.e. cardioprotective effect of brief transient non-lethal ischemia). This a

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Lineare und quadratische Kongruenzen,rised in both animals and in the human, and appears to involve adenosine receptor activation, protein kinase C mediation and the possible opening of ATP-dependent-potassium (K.) channels. If the exact mechanism of preconditioning in man could be determined, then it might be possible to develop preco

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Lineare und quadratische Kongruenzen,hen phosphorylates K. channels and the phosphorylation makes the channel open more readily during the second ischemia. The protective effect of adenosine has been linked to K. channel opening: The protection induced by preconditioning, adenosine receptor agonists and PKC activators can be eliminated