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Titlebook: Acute Leukemias VI; Prognostic Factors a T. Büchner,G. Schellong,B. Wörmann Conference proceedings 1997 Springer-Verlag Berlin Heidelberg 1

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Protection Against Snow Avalanche Hazardsion arising from these findings is whether cytogenetic investigations may not be sensitive enough to detect the malignant cell clone at certain stages of the disease. We present three cases with aberrant karyotypes at initial diagnosis prior to therapy and with normal cytogenetic results in hematolo
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Shiva P. Pudasaini,Kolumban Huttera complementation strategy we used recently to define the signaling pathways activated by the Bcr-Abl tyrosine kinase. Point mutations were generated in domains of Bcr-Abl that may play a role in connecting its tyrosine kinase signal to downstream effector molecules. Hyperexpression of c-Myc, a gene
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Shiva P. Pudasaini,Kolumban Huttertic leukemia. Recently, the . gene (pl6., MTS I, . has been proposed as a candidate tumor suppressor gene because it is frequently deleted in cell lines derived from multiple tumor types. We examined 18 leukemia-derived cell lines (13 T-ALL, 3 B-cell-precursor ALL,1 AML,1 CML-blast crisis) by polyme
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https://doi.org/10.1007/978-3-540-32687-8 cases. In contrast, oligoclonality as deduced from diversity of the TcR-d gene rearrangements of the immature types has not been reported so far. In 4 of 20 cases of childhood BCP-ALL we detected oligoclonality characterized by the coexistence of different junctional regions varying in size by 3-25
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Haematology and Blood Transfusion‘ Hämatologie und Bluttransfusionhttp://image.papertrans.cn/a/image/144388.jpg
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