Titlebook: Whole Energy Systems; Bridging the Gap via Vahid Vahidinasab,Behnam Mohammadi-Ivatloo Book 2022 The Editor(s) (if applicable) and The Autho

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书目名称Whole Energy Systems影响因子(影响力)




书目名称Whole Energy Systems影响因子(影响力)学科排名




书目名称Whole Energy Systems网络公开度




书目名称Whole Energy Systems网络公开度学科排名




书目名称Whole Energy Systems被引频次




书目名称Whole Energy Systems被引频次学科排名




书目名称Whole Energy Systems年度引用




书目名称Whole Energy Systems年度引用学科排名




书目名称Whole Energy Systems读者反馈




书目名称Whole Energy Systems读者反馈学科排名

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Omid Sadeghian,Arman Oshnoei,Behnam Mohammadi-Ivatloo,Vahid Vahidinasabing fetus is particularly vulnerable to adverse influences in the environment. . Studies of malformations at birth remained largely in the hands of paediatricians who, for instance, quickly established the con­ nection between rubella infection in early pregnancy and the birth of severely affected i

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Vahid Shabazbegian,Hossein Ameli,Mohammad Taghi Amelicritical technical challenges in D2D communications are discussed, and D2D standardization activities in 3GPP are provided. Topics range from proximity discovery and mode selection, to resource management. The authors investigate proximity detection solutions for enabling direct user equipment commu

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Mohammad Mehdi Davary,Mohammad Taghi Ameli,Hossein Ameliate estimates for the present day biodiversity to help with Carbonate depositional systems in the Paleozoic geologic time represent fewer studies in paleoecological interactions than the siliciclastic systems. To evaluate this difference, the paleontology of the Middle Devonian Dundee Formation in O

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Masoud Agabalaye-Rahvar,Amin Mansour-Saatloo,Mohammad Amin Mirazaei,Behnam Mohammadi-Ivatloo,Kazem Zcessive fat accumulation and is defined by a body mass index ≥30 kg/m.. Obesity increases the risk for metabolic and cardiovascular diseases, musculoskeletal disorders, some types of cancer, pulmonary diseases, and psychological diseases. A doubling of the obesity prevalence since the 1980s may be c

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Amirhossein Khalili-Garakani,Leila Samiee,Kazem Kashefi/or antiapoptotic effects of other growth factors, cytokines, or transforming oncogenes (.). These agonists all stimulate Akt/PKB by promoting its phosphorylation on two regulatory residues (e.g., S473 and T308 for the Akt1 isoform), an event dependent on the prior activation of a signaling pathway