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Titlebook: Was Sie schon immer über Sucht wissen wollten; Werner Gross Book 2016 Springer-Verlag Berlin Heidelberg 2016 Abhängigkeit.Alkohol.Co-Abhän

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Werner Grossubsequently, the emphasis was shifted to mutation as a cellular process in which the primary lesion was extensively modified by enzymatic DNA-repair systems constitutively present in the cell (Auerbach 1976). Finally, over the past 15 years, considerable interest has been focused on inducible cellul
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Werner Grossrcomas stop proliferation after 3–4 in vitro passages, not allowing sufficient material for molecular analyses by in vitro preparation to be obtained (for PSA, see Stern et al. 1990), One alternative to overcome the problem of replicative senescence (Hayflick and Moorhead 1961) of tumor cells in cul
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Werner Grossxcitement about deciphering the composition of the centromere DNA..Active as well as inactive centromeres have similar morphology; the latter appear as bonafied primary constrictions and propagate faithfully. However, when hybridized to the mouse minor satellite DNA some, but not all, inactive centr
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Werner Grossorder to provide clues to the mechanisms of cytological and molecular evolution of patterns, A model is presented to explain the identified rules..Within a heterochromatin-rich complement, the shorter chromosomes (or chromosome arms) tend to have large telomeric C-bands whereas the longer chromosome
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Werner Grossdegenerative disease of the central nervous system, characterized by cerebellar ataxia and trembling, called “Kuru” by the New Guineans who suffer from it. This disease, restricted to some 12,000 natives of the Fore linguistic group indigenous to the Eastern Highlands of New Guinea and to their imme
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Werner Grossastic-transformed stem cell [10]. In more than 90% of cases, an indicative reciprocal translocation t(9.22), known as the “Philadelphia chromosome,” is present [39]. The observation that natural interferon-alpha (IFN-α) and fibroblast interferon (IFN-β) caused inhibitory effects on the in vitro grow
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Werner Grossd and his associates [1] developed the concept that during forced expiration lateral pressures at points within the airways become equal to pleural pressure and that the pressure driving flow from the alveoli to these equal pressure points approximates the static recoil pressure of the lung. This me
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