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Titlebook: 2nd International Adalat® Symposium; New Therapy of Ische Wilhelm Lochner (President),Wolfgang Braasch,Günth Conference proceedings 1975 Sp

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https://doi.org/10.1007/978-3-7908-2649-4F. . [3] demonstrated that nifedipine decreases the contractility and oxygen consumption of the isolated papillary muscle of guinea pig. These results were confirmed by R. . [5], who demonstrated negative inotropic effects and a decreasing action on the oxygen consumption of nifedipine in the isolated Langendorff-guinea pig heart preparation.
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https://doi.org/10.1007/978-1-4020-9567-2As long as there is no causal therapy of coronary disease, treatment of coronary insufficiency can only be symptomatic and prophylactic. Animal experiments by . . [4] demonstrated that nifedipine causes a rapid, long-lasting increase of coronary blood flow.
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A Theory of Forced Labour MigrationThe pharmacokinetic investigations were carried out at the 2nd University Clinic, Düsseldorf, and the results were determined in the Isotope Institute of Bayer, Wuppertal-Elberfeld, by Drs. Duhm, Maul, Medenwald, Patschke and Wegner.
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https://doi.org/10.1007/978-3-642-66241-6Adalat; angina pectoris; chemistry; circulation; coronary artery disease; diseases; heart; hemodynamics; the
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978-3-540-07471-7Springer-Verlag Berlin Heidelberg 1975
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https://doi.org/10.1007/b138812 this symposium we shall not deal with the treatment or prevention of arteriosclerosis as the main cause of ischemic heart disease. We shall discuss rather the improvement of the relationship between oxygen supply and oxygen consumption of the heart, both in the acute state of angina and as long-ter
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https://doi.org/10.1007/978-3-7091-4313-1 of coronary and myocardial effects—on the one hand, and extracardial effects on the systemic and pulmonary circulation on the other. To a certain extent both components may improve the disproportion between oxygen supply and oxygen demand of the myocardium in coronary artery or primary myocardial d
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Wave Motions in a Laminated Medium place simultaneously with liberation of Ca from certain endoplasmic stores. The rapid rise in free intracellular Ca during excitation initiates the splitting of ATP by the Ca-dependent ATPase of the myofibrils so that phosphate-bond energy is transformed into mechanical work. This means, in other w
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