Titlebook: Vitreous; in Health and Diseas J. Sebag Book 2014 Springer Science+Business Media New York 2014 biochemistry.ophthalmology.pathology.pharma

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I.C. Hereditary Vitreo-Retinopathiesalso in articular and hyaline cartilage. The phenotypes can be variable but affected patients classically exhibit high degrees of congenital myopia and visual loss from retinal detachment with varying degrees of associated hearing loss, arthropathy, and orofacial abnormalities.

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II.B. Myopic Vitreopathyated from the potentially blinding associations of pathological myopia with an arbitrary refractive error of −6.0D [4]. However it is becoming clear that there is no threshold effect and that common myopia of all levels contributes to risks of uncorrectable visual loss such as cataract, glaucoma, retinal detachment, and maculopathy [5, 6].

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III.C. Pathology of Vitreomaculopathiesither be tangential to the retinal surface or perpendicular (axial). In reality, both pathologies are frequently found in combination, forming characteristic pathologic features of clinical diseases such as macular pucker, macular hole, and vitreomacular traction syndrome.

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II.C. Vitreous Aging and Posterior Vitreous Detachments: in the gel vitreous body and at the vitreoretinal interface. Although remarkably common and usually harmless, PVD is the single most important factor underlying pathology that results from the aging process in the vitreous [see chapter .. Anomalous posterior vitreous detachment and vitreoschisis].

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III.B. Anomalous Posterior Vitreous Detachment and Vitreoschisisa better understanding of vitreoretinal disease pathogenesis, improve diagnostic acumen, and provide new directions for therapeutic approaches that will ultimately lead to effective preventative strategies.

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