Titlebook: Vitamin D; Physiology, Molecula Michael F. Holick Book 19991st edition Humana Press 1999 Absorption.Calcium.Metabolite.Public Health.Vitami

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Metabolism and Catabolism of Vitamin D, Its Metabolites, and Clinically Relevant Analogs, functional complex and binding to the vitamin D binding protein (DBP) are probably key elements in dictating the differences in the actions of so-called calcemic and noncalcemic vitamin D analogs. Therefore, from the perspective of its historical significance and relevance, it seems entirely logica

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The Enzymes Responsible for Metabolizing Vitamin D,. by 25-hydroxyvitamin D. 24-hydroxylase (24-hydroxylase), existing in proximal convoluted tubules of the kidney .. The two kidney hydroxylases are induced in the reciprocal calcium statuses. lα-Hydroxylase activity is enhanced in a calcium- and/or 1,25(OH).D.depleted status, whereas that of 24-hydr

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Molecular Biology of the Vitamin D Receptor,es skeletal calcium by ensuring that adequate absorption of dietary calcium takes place. In addition to this calciotropic role, vitamin D functions in a plethora of cellular actions, perhaps the most fundamental of which is cellular differentiation .. In skeletal tissue, 1,25(OH).D. increases osteoc

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Molecular Recognition and Structure-Activity Relations in Vitamin D-Binding Protein and Vitamin D R functions manifest (.) (Fig. 1). In addition to vitamin D. (synthesized in the skin), another chemical form of vitamin D. (called vitamin D., which is obtained primarily from diet and vitamin D supplements) and all of its metabolites exist in nature. Vitamin D. is metabolized to 25-hydroxyvitamin D

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Biologic and Molecular Effects of Vitamin D on Bone,ty of cells of the osteoblast lineage, which form the mineralized bone matrix and cells of the osteoclast lineage, which resorb the mineralized bone (Fig. 1). Vitamin D exerts its effects on these cells by modulating the transcription of a broad spectrum of genes related to these bone cell phenotype

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Regulation of Parathyroid Hormone Gene Expression and Secretion by Vitamin D,1,25(OH).D. was due to the increased intestinal absorption of calcium and the increased levels of serum calcium, leading to a decrease in parathyroid hormone (PTH) secretion. This is certainly correct, but it is only part of the story. It was later shown by a number of in vitro and in vivo studies t

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Book 19991st edition as in the choice of chapter authors. The international perspective, especially with regard to public health initiatives, is emphasized where appropriate. The editors, whose training is both research and practice oriented, have the opportunity to develop a primary objective for their book, define th

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2628-197X e appropriate. The editors, whose training is both research and practice oriented, have the opportunity to develop a primary objective for their book, define th978-1-4757-2861-3Series ISSN 2628-197X Series E-ISSN 2628-1961

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