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Titlebook: Viruses and Human Cancer; From Basic Science t T.-C. Wu,Mei-Hwei Chang,Kuan-Teh Jeang Book 2021Latest edition Springer Nature Switzerland A

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书目名称Viruses and Human Cancer
副标题From Basic Science t
编辑T.-C. Wu,Mei-Hwei Chang,Kuan-Teh Jeang
视频video
概述Provides a comprehensive overview of the basic and clinical aspects of oncogenic viruses and associated human cancers.Covers viral infection, virus-induced inflammation and tissue injuries, viral epid
丛书名称Recent Results in Cancer Research
图书封面Titlebook: Viruses and Human Cancer; From Basic Science t T.-C. Wu,Mei-Hwei Chang,Kuan-Teh Jeang Book 2021Latest edition Springer Nature Switzerland A
描述.This book, in a new, extensively updated edition, covers viral infection, virus-induced inflammation and tissue injuries, viral epidemiology, oncogenic mechanisms, and current and emerging preventive and therapeutic strategies in detail. Readers will also find information on the individual aspects of a number of oncogenic viruses, including hepatitis B, hepatitis C, human papillomavirus, Epstein–Barr virus, human T-cell lymphotropic virus, Kaposi sarcoma-associated herpes, and Merkel cell polyomavirus, as well as associated human cancers. .The book will benefit all those who are seeking a comprehensive, up-to-date overview of the basic and clinical aspects of oncogenic viruses and associated human cancers. Following its original publication in 2014, the first edition of this book quickly became an influential text in the field. This second edition duly reflects the significant advances in knowledge and research that have been achieved in the years since. .
出版日期Book 2021Latest edition
关键词Viruses and Human Cancer; Viruses Infection; Inflammation; Cancer Prevention; Oncogenic Role of Hepatiti
版次2
doihttps://doi.org/10.1007/978-3-030-57362-1
isbn_softcover978-3-030-57364-5
isbn_ebook978-3-030-57362-1Series ISSN 0080-0015 Series E-ISSN 2197-6767
issn_series 0080-0015
copyrightSpringer Nature Switzerland AG 2021
The information of publication is updating

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Mechanisms of Hepatitis B Virus-Induced Hepatocarcinogenesis,ly infected by this virus, resulting in nearly 1 million deaths every year. Many of these patients die from severe liver diseases, including HCC. HBV may induce HCC through the induction of chronic liver inflammation, which can cause oxidative stress and DNA damage. However, many studies also indica
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Prevention of Hepatitis B Virus Infection and Liver Cancer,of HCC in the world. Prevention is the best way to control cancer. There are three levels of liver cancer prevention, i.e., primary prevention by HBV vaccination targeting the general population starting from birth dose, secondary prevention by antiviral agent for high-risk subjects with chronic HBV
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The Oncogenic Role of Hepatitis C Virus,nly inflammation and subsequent fibrosis but also HCV itself are associated with hepatocarcinogenesis. To date, studies using transgenic mouse and cell-culture models, in which HCV proteins are expressed, indicate the direct pathogenicity of HCV, including oncogenic activity. In particular, the core
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High-Risk Human Papillomaviruses and DNA Repair,s oral cancers. High-risk HPVs are responsible for nearly half of all virally induced cancers. Viral replication and amplification are intimately linked to the stratified epithelium differentiation program. The E6 and E7 proteins contribute to the development of cancers in HPV positive individuals b
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,Epstein–Barr Virus-Associated Post-transplant Lymphoproliferative Disease,d T cell lymphomas, epithelial, and mesenchymal tumors. The virus is ubiquitous, transmitted in saliva, and not usually associated with the development of malignancy. PTLD is usually associated with EBV when it occurs soon after the transplant. Measurement of viral DNA in blood, especially plasma, m
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