Titlebook: Vascular Endothelium; Pharmacologic and Ge John D. Catravas,Allan D. Callow,Una S. Ryan Book 1998 Springer Science+Business Media New York

Waterproof

书目名称Vascular Endothelium影响因子(影响力)




书目名称Vascular Endothelium影响因子(影响力)学科排名




书目名称Vascular Endothelium网络公开度




书目名称Vascular Endothelium网络公开度学科排名




书目名称Vascular Endothelium被引频次




书目名称Vascular Endothelium被引频次学科排名




书目名称Vascular Endothelium年度引用




书目名称Vascular Endothelium年度引用学科排名




书目名称Vascular Endothelium读者反馈




书目名称Vascular Endothelium读者反馈学科排名

DIS

monopoly

Mechanisms of Endothelial Injury and Repairoducts such as the reactive oxygen species (ROS), proteases and the already mentioned cytokines. The effects of oxygen radicals will be discussed more specifically because they represent a well studied model of endothelial injury. They are also associated with other important factors implicated in microvascular damage.

Hemiparesis

Cytokine Regulation of Endothelial Cell Functionnd function of white blood cells require an intimate relationship with vascular EC. Cytokines are mediators of these complex bi-directional interactions between leukocytes and vascular elements (for review see Mantovani and Dejana, 1989, Mantovani, Bussolino, and Dejana, 1992).

Mitigate

Mechanisms of Endothelial Injury and Repairspiratory distress syndrome being a paradigm of such a complex situation where toxic and inflammatory elements interact. The role of cytokines has received prominent attention and their involvement in the expression of adhesion molecules has emphasized the importance of the intricate relationship be

秘方药

MILL

仲裁者

Control of iNOS Expression in Rat Aortic Smooth Muscle Cellse NO synthase (iNOS or type II NOS) in a variety of cells, including vascular endothelial and smooth muscle cells.. This enzyme also uses L-arginine as substrate and is sensitive to inhibitory L-arginine analogues; furthermore, it does not appear to differ significantly from ecNOS on the basis of co

seduce

埋伏

Insertion/Deletion (I/D) Polymorphism in the Angiotensin Converting Enzyme (ACE) Gene the report by Cambien and his co-workers (Cambien et al., 1992) of association between the D allele and coronary heart disease (CHD), particularly in CHD patients without classical risk factors. In the following, this observation by Cambien’s group will be compared with observations in our group an