Titlebook: Systems Biology of Alzheimer‘s Disease; Juan I. Castrillo,Stephen G. Oliver Book 2016 Springer Science+Business Media New York 2016 Alzhei

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书目名称Systems Biology of Alzheimer‘s Disease影响因子(影响力)




书目名称Systems Biology of Alzheimer‘s Disease影响因子(影响力)学科排名




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书目名称Systems Biology of Alzheimer‘s Disease网络公开度学科排名




书目名称Systems Biology of Alzheimer‘s Disease被引频次




书目名称Systems Biology of Alzheimer‘s Disease被引频次学科排名




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书目名称Systems Biology of Alzheimer‘s Disease年度引用学科排名




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书目名称Systems Biology of Alzheimer‘s Disease读者反馈学科排名

frivolous

Application of Systems Theory in Longitudinal Studies on the Origin and Progression of Alzheimer’s D alzheimer’s disease (EOAD) could serve as a linear model to study the pathogenesis of sporadic late-onset alzheimer’s disease (LOAD). Now there is growing evidence to suggest that such reductionism may not be warranted; these suppositions are not adequate to explain the molecular complexities of LO

Carbon-Monoxide

Kidnap

极大痛苦

Advanced Assay Monitoring APP-Carboxyl-Terminal Fragments as Markers of APP Processing in Alzheimer ecretase cleavage and is the direct precursor of amyloid beta (Aβ). Here we describe a method for the quantification of C99. The amount of C99 is an indicative value of the amyloid pathology in an Alzheimer’s disease (AD) model, and could be used as a marker to study AD progression in comprehensive

FISC

厚脸皮

Protocols for Monitoring the Development of Tau Pathology in Alzheimer’s Diseasey known as tauopathies. Development of tau pathology is associated with progressive neuronal loss and cognitive decline. In the brains of AD patients, tau pathology spreads following a predictable, anatomically defined progression pattern that can be followed by immunohistochemistry looking at brain

altruism

枕垫

Advanced Mitochondrial Respiration Assay for Evaluation of Mitochondrial Dysfunction in Alzheimer’s tau) in the brain, but the underlying mechanisms of the disease are still partially unclear. A growing body of evidence supports mitochondrial dysfunction as a prominent and early, chronic oxidative stress-associated event that contributes to synaptic abnormalities, and, ultimately, selective neuro

忙碌

Analysis of Microglial Proliferation in Alzheimer’s Diseaseormation is available for specific neurodegenerative disorders, particularly for Alzheimer’s disease (AD). Determining the degree of local proliferation will not only open avenues into understanding the dynamics of microglial proliferation, but also provide an effective target to design strategies w