Titlebook: Studies on Periodontal Disease; Daisuke Ekuni,Maurizio Battino,Edward E. Putnins Book 2014 Springer Science+Business Media New York 2014 C

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The Role of p53 in Carcinogenesis and Apoptosis in Oral Tissues well as host factors increase ROS. The volatile sulfur compounds in the oral cavity, especially oral malodorous compounds, are the most probable producers of ROS. The functions of p53 involve causing p53-dependent apoptosis, repairing DNA-strand errors and arresting the cell cycle for DNA repair. A

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Physiological Roles of a Periodontopathic Bacterial Membrane-Bound Quinol Peroxidaseof ., a bacterial species associated with a variety of human infections, including localized aggressive periodontitis (LAP). The cytoplasmic membrane of . exhibited QPO-dependent peroxidase activity in the presence of NADH or succinate, known physiological substrates of the respiratory chain. QPO is

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The Inflammation as the Connecting Link Between Systemic Diseases and Periodontal Diseases of the connecting link among them. This chapter shows the recent advances about systemic inflammatory markers in periodontitis patients, the effect of periodontal therapy over systemic inflammatory markers, and some putative pathogenic mechanisms that could be shared with other conditions. C-reacti

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Periodontitis and Atherosclerosiscate that reactive oxygen and nitrogen species have a causal role in atherosclerosis. Periodontitis is one of the most widespread inflammatory diseases, and it induces excessive production of reactive oxygen species in periodontal lesions. Previous studies have suggested that there is an association

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