Titlebook: Strategies in Clinical Hematology; Rudolf Gross,Klaus-Peter Hellriegel Conference proceedings 1979 Springer-Verlag Berlin Heidelberg 1979

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,Childhood Acute Lymphocytic Leukemia — A Model for Therapeutic Strategies in Hemopoietic Neoplasia,which physicians have no control. The central question is, why has childhood ALL responded to treatment much better than acute myelocytic leukemia (AML) at any age, or ALL in adults? Several factors contribute to this difference:

钳子

Leukemias and Lymphomas Associated with the Use of Cytotoxic and Immunosuppressive Drugs, of which have profound effects upon the bone marrow, lymphoid tissues, and the circulating blood. Most changes, such as leukopenia, thrombocytopenia, or anemia are usually reversible when treatment is stopped. However, sometimes progressive myeloproliferative or lymphoproliferative disorders may oc

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含铁

Immunologic Markers for Classification of Leukemias and Non-Hodgkin Lymphomas,monotonous acute, chronic, and even (to a certain extent) undifferentiated leukemias can now be classified into several subgroups using various immunologic test systems. A correlation of the immunologic phenotype with cell count, sex, evolution of the disease etc. has been found for T leukemias [2,

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Can Remission Duration Be Prolonged in Acute Myeloid Leukaemia?,-cell population to zero, because it seems probable, though not certain, that the leukaemic blast cells constitute a clone, genetically isolated from the normal stem-cell population from which they arose, and that their selective destruction would leave a normal stem-cell population with a relativel

敲诈

,Advances in the Therapy of Non-Hodgkin’s Lymphoma, other factors related to prognosis have been critically revised. New therapeutic strategies have been developed on the basis of successful simultaneous application of antitumor drugs. New agents have been synthetized which proved to be useful as second and third choice treatment compounds. Within t

经典

Advances in Hereditary Red Cell Enzyme Abnormalities,than 20 different red cell enzymopathies with red cell dysfunction have been described. In addition several metabolic diseases can be diagnosed through detection of a specific enzyme defect in red cells, but do not alter red cell function and viability or normal oxygen carriage and delivery to the t

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