Titlebook: Spinal Cord Monitoring; Basic Principles, Re Erik Stålberg,Hari Shanker Sharma,Yngve Olsson Book 1998 Springer-Verlag Wien 1998 Neurology.e

Orchiectomy

厚颜

abnegate

chemical-peel

大范围流行

A combination of microdialysis, electrophysiology and histology for exploring secondary damage upon uma by secondary destructive processes that include release of agents that kill neurons, post injury ischemia, edema, inflammation, and oxidative damage. Agents suspected of causing secondary damage include excitatory neurotransmitters, free fatty acids, neuropeptides and free radicals. Oxidative da

抛射物

Spinal cord bioelectrical activity, edema and cell injury following a focal trauma to the rat spinaltor and Fehlings, 1991 ; Faden and Salzman, 1992; Faden, 1993a; Schwab and Bartholdi, 1996). The prevalence of spinal cord injury is about 30 to 50 cases per million population per year in the United States of America which is quite comparable to Europe and other continents (Tator and Edmonds, 1979;

展览

Spinal cord blood flow and evoked potentials as outcome measures for experimental spinal cord injury one or more additional damaging processes initiated by the primary injury (Collins, 1983; Sandier and Tator, 1976a,b). Many pathophysiological processes are involved in the secondary injury mechanisms including vascular, electrolyte and biochemical changes, edema and loss of energy metabolism (Tato

乐章

Spinal cord blood flow and metabolism in transient spinal ischemia resulting from critical reduction of substrate (glucose/oxygen) delivery and corresponding release of active factors (excitatory amino acids, prostaglandins, oxygen free radicals etc.) into the spinal extracellular space.

帽子

现任者