Titlebook: Shock, Sepsis, and Organ Failure — Nitric Oxide; Fourth Wiggers Berna Günther Schlag,Heinz Redl Conference proceedings 1995 Springer-Verlag

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The Role of Nitric Oxide in Hemorrhagic Shock and Trauma, and immune components such as catecholamines, Cortisol, multiple cytokines, complement, and products of arachidonic acid metabolism. Studies which differentiate the roles played by individual mediators continue to be central to our current understanding of the injured patient.

EWER

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Inhibition of Induction or Activity of Nitric Oxide Synthase: Novel Approaches for the Therapy of C (a) improved life-support technology which keeps intensive care patients with high risk of infection alive for prolonged periods, (b) the prevalence of immunocompromised patients due to an increased incidence of acquired immunodeficiency syndrome or due to chemotherapy and immunotherapy, and (c) th

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Inhibition of Nitric Oxide Synthase During Sepsis and Endotoxemia May Be Detrimental,O synthase activity during sepsis and endotoxemia may be detrimental. Thus, evidence of increased mortality, liver and lung injury, and decreased intestinal integrity has been reported following treatment with NO synthase inhibitors during sepsis and endotoxemia.

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Effect of Nitrosylated Albumin in the Isolated Rabbit Heart,hy the reaction pathway of (endothelium-derived) NO should be restricted to inactivation by oxygen or by soluble guanylyl cyclase. The NO radical is in principle capable of oxidizing many important molecules such as compounds containing thiol groups, heme groups, iron-sulfur centers, (primary) amine