Titlebook: Secondary Hypertension; Clinical Presentatio George A. Mansoor Book 2004 Springer Science+Business Media New York 2004 drugs.endocrinology.

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Diagnostic Evaluation for Patients With Renovascular Hypertension10 and 45% of those in a hypertension subspecialty practice .. Thus, routine screening is neither cost effective nor efficient; screening should be performed only on those patients with historical and physical findings, which raise suspicion for renovascular disease. Additionally, renal artery steno

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Medical Management of Renovascular Hypertensionsity, and complexity of treatment can vary widely. In a young woman with the abrupt onset of hypertension and an abdominal bruit, an astute clinician will suspect renovascular hypertension caused by fibromuscular dysplasia (FMD). With early diagnosis, the course of drug therapy is often short (weeks

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Primary Reninism or Renin-Secreting Tumors liver is cleaved by active renin, which comes primarily from the juxtaglomerular (JG) cells of the renal afferent arteriole, to release the decapeptide angiotensin I (Ang I). Ang I is in turn cleaved by endothelial cell-bound angiotensin-converting enzyme (ACE) to produce the vasoactive peptide ang

thrombus

Primary AldosteronismA), increased plasma aldosterone concentration (PAC), and unsuppressible aldosterone levels in the blood or urine. Prevalence estimates for primary aldosteronism when hypokalemia was one of the diagnostic criteria varied from 0.05 to 2% of the population with hypertension. However, with the recognit

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Medical Management of Primary Aldosteronism or hyperplasia). Medical therapy is used to reverse the metabolic and hemodynamic sequelae of aldosterone excess before surgery and in the long-term management of patients who are not surgical candidates. The latter include patients with bilateral disease who require bilateral adrenalectomy or who

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