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Titlebook: Scheitern; Aspekte eines sozial Matthias Junge,Götz Lechner Book 2004 VS Verlag für Sozialwissenschaften/GWV Fachverlage GmbH, Wiesbaden 20

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inical diagnosis of AD very simple and clear-cut in most of the cases. In contrast, the underlying mechanisms leading to the manifestation of AD are more than complex and consist of genetic components combined with various deficiencies on the level of innate and adaptive immune mechanisms. Challenge
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Matthias Junge,Götz Lechnerf the International Immune-Mediated Diseases: From Theory to Therapy (IMD) Congress is to bring the best world immunologists and clinicians to Moscow to participate in plenary sessions, symposia and educational workshops designed to expose basic and clinical immunologist as well as practicing clinic
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Götz Lechner antigens in the thymus has been implicated in T cell tolerance and autoimmunity. During the last years, it has been shown that medullary thymic epithelial cells (mTECs) are the unique cell type expressing a diverse range of tissue-specific antigens. Promiscuous gene expression is a cell autonomous
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Klaus Feldmann myelin sheath generated by Schwann cells, whereas others are unmyelinated. Peripheral nerves serve different motor, sensory, and autonomic functions. The term peripheral neuropathy is usually used to describe symmetric and universal damage to adjacent nerves. The damage and clinical manifestations
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Annette Spellerbergogy, which differentiates between acquired or inherited forms. Among the acquired and hereditary neuropathies further subdivision has been proposed. Acquired and hereditary neuropathies are classified according to the course as acute or chronic. Acquired and hereditary neuropathies may be accompanie
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Heike Solgaie durch Autoantikörper (7SIgG-Globuline) verursacht wird. Das klinische Bild der durch den Thrombozytenmangel verursachten hämorrhagischen Diathese ist sehr variabel. Der Altersgipfel der haemorrhagischen Diathese ist sehr variabel. Der Altersgipfel dieser Erkrankung liegt im führen Erwachsenenalte
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Corinna Onnen-Isemannie durch Autoantikörper (7SIgG-Globuline) verursacht wird. Das klinische Bild der durch den Thrombozytenmangel verursachten hämorrhagischen Diathese ist sehr variabel. Der Altersgipfel der haemorrhagischen Diathese ist sehr variabel. Der Altersgipfel dieser Erkrankung liegt im führen Erwachsenenalte
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